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Results

平成23年度研究成果

（高久洋）

  1. Nishitsuji, H., Yokoyama, M., Sato, H., Yamauchi, S., Takaku, H., Identification of amino acid residues in HIV-1 reverse transcriptase that are critical for the proteolytic processing of Gag-Pol precursors, FEBS Lett. 585, 3372-3377 (2011).
  2. Sugiyama, R., Naganuma, H., Nishitsuji, H., Takaku, H., Human immunodeficiency virus-1 Nef suppresses Hsp70-mediated Tat activation, FEBS Lett. 585, 3367-3371 (2011).
  3. Noguchi, K., Ishitu, Y., Takaku, H., Evaluating target silencing by short hairpin RNA mediated by the group I intron in cultured mammalian cells, BMC Biotechnol. 11, 79 (2011).
  4. Sugiyama, R., Nishitsuji, H., Furukawa, A., Katahira, M., Habu, Y., Takeuchi, H., Ryo, A., Takaku, H., Heat shock protein 70 inhibits HIV-1 Vif-mediated ubiquitination and degradation of APOBEC3G, J. Biol. Chem. 286, 10051-10057 (2011).

（下遠野邦忠）

  1. Shimizu Y, Hishiki T, Ujino S, Sugiyama K, Funmi K, Shimotohno K. Lipoprotein components associated with hepatitis C virus is essential for virus infectivity, Current Opinion in Virology, 1, 19-26, 2011.
  2. Aly HH, Oshiumi H, Shime H, Matsumoto M, Wakita T, Shimotohno K, Seya T. Development of mouse hepatocyte lines permissive for hepatitis C virus (HCV). PLoS One, 6, e21284, 2011.
  3. Onomoto K, Morimoto S, Kawaguchi T, Toyoda H, Tanaka M, Kuroda M, Uno K, Kumada T, Matsuda, F, Shimotohno K, Fujita T, Murakami Y., Dysregulation of IFN system can lead to poor response to pegylated interferon and ribavirin therapy in chronic hepatitis C, PLoS One, 6, e19799, 2011.
  4. Morohashi K, Sahara H, Watashi K, Iwabata K, Sunoki T, Kuramochi K, Takakusagi K, Miyashita H, Sato N, Tanabe A, Shimotohno K, Kobayashi S, Sakaguchi K, Sugawara F. Cyclosporin A associated helicase-like protein facilitates the association of hepatitis C virus RNA polymerase with its cellular cyclophilin B, PLoS One, 6, e18285. 2011.
  5. Murakami Y, Toyoda H, Tanaka M, Kuroda M, Harada Y, Matsuda F, Tajima A, Kosaka N, Ochiya T, Shimotohno K. The Progression of Liver Fibrosis Is Related with Overexpression of the miR-199 and 200 Families, PLoS One, 6, e16081, 2011.
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